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The first studies on heteromers of the D 4 R polymorphic variants, based on BRET and co-immunoprecipitation techniques in transfected mammalian cells, suggested that the D 4.7 R establishes weaker intermolecular and functional interactions with the D 2 R (both isoforms, D 2L R and D 2S R) than the D 4.4 R ( 2 Rs with D 4.4 Rs, but not D 4.7 Rs, led to an increase in MAPK signaling ( 4.7 R gain of function

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